Critica de la neurosis de angustia by Freud Sigmund

By Freud Sigmund

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1990; Voutsinas 1987). It is discussed whether a massive HIV coinfection in PML lesions contributes to the occasionally necrotizing character of these foci (Budka 1990; Schmidbauer et al. 1990). In cases with space-occupying PML lesions, a coinciding toxoplasmosis or lymphoma must be suspected (Tosch et al. 1990). Guilleux et al. (1986) described the MRI findings in PML seen in HIV-negative patients. They observed an increased Tl relaxation time (low signal intensity on Tl-WI), shortened TI relaxation time (high signal intensity on TI-WI), involvement of the white matter, asymmetrical distribution, no predilection for the periventricular regions, and no mass effect (Fig.

1991). 40 a c Chapter 2 Diagnostic Imaging of Intracranial Manifestations of AIDS b CNS Infections 41 e ~ f ... Fig. 16 a-f. MRI appearance of progressive diffuse leukoencephalopathy. The pattern has basically the same distribution as known from CT. As expected, the affected frontal white matter is hyperintense on TZ-WI (a--e) and hypointense on Tl-WI (f). Additional sites of demyelination, for example, in the occipital lobe (a,b) or along the internal capsule (d) are frequently detected by TZ-Wl.

1989; Ramsey and Geremia 1988). In patients with mUltiple lesions several patterns may coexist (Post et al. 1983). The enhancing properties of the lesions are, on the one hand, to some extent dependent on the immunological reactivity of the host. A strong immune reaction leads to a severe disturbance of the bloodbrain barrier and to massive contrast enhancement (Gaston et al. 1985). On the other hand, contrast-enhancement indicates the acuteness of a lesion during the course of the disease. Both should regress under antitoxoplasmotic chemotherapy.

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