Bacterial Virulence by P. Sansonetti

By P. Sansonetti

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Interestingly, it is rarely associated with clinical symptoms, unless the patient also suffers from diabetes mellitus. In this case, the diabetic patient may develop disseminated candidiasis or other fungal infections. In this last case, the diabetic patient can develop disseminated candidiasis or other fungal infections. In vitro, MPO deficient neutrophils kill C. albicans and Aspergillus fumigatus hyphae poorly. G6PD deficiency is due to mutations in G6PD, and affects mainly erythrocytes. Only in rare cases do these mutations lead to extremely low levels of G6PD in erythrocytes and neutrophils, inducing chronic and severe hemolytic anemia and CGD-like symptoms [20].

Upon stimulation, the secretory vesicles are mobilized to the plasma membrane and provide a new source of receptors and other functional proteins. The exocytosis of these vesicles is important during rolling on an activated endothelium to establish firm adhesion with β2-integrins [11, 12]. 2 Mobilization and Fusion Granules are spread throughout the cytoplasm and are recruited for fusion at either the plasma membrane or the phagosome. The precise mechanism of granule mobilization and delivery is not completely understood.

Care, 7 (4), 291–307. H. (2007) Selectin inhibitors and their proposed role in ischemia and reperfusion. J. Invest. , 20 (2), 71–85. 4 Ley, K. (2003) Arrest chemokines. Microcirculation, 10 (3–4), 289–295. , and Ley, K. (2008) Mechanisms and consequences of 6 7 8 9 neutrophil interaction with the endothelium. Am. J. , 172 (1), 1–7. , and Sandig, M. (2001) Neutrophil diapedesis: paracellular or transcellular? News Physiol. , 16, 15–19. , et al. (2003) Phagocytosis by neutrophils. Microbes. , 5 (14), 1299–1306.

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